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Detoxification, methylation and anti-oxidants continue to play an important role in the biomedical treatment of autism and other chronic medical conditions

These findings were not attributed to variations in food intake, which implies that the observed results were mediated by alterations in metabolic processes [1]

However, ferristatin-1 can specifically prevent ferroptosis induced by erastin, but mitochondria are not involved in the function of ferristatin-1, suggesting that mitochondria may not be necessary for ferroptosis [46]

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