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We identify melatonin as a previously unrecognized regulator of ILC2 metabolism and function

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Astroglial atrophy and loss of function A widespread class of astrocytic changes observed in many neurological diseases and in the majority of neuropsychiatric disorders are represented by structural atrophy and functional asthenia and manifested in the loss of key homoeostatic functions, such as for example glutamate clearance

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113,114,115,116 Activation of these receptors initiates an inflammatory signaling cascade resulting in the secretion of pro-inflammatory cytokines and chemokines