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Nausea results primarily from delayed gastric emptying and central nervous system effects

[11] [4] Preclinical studies have suggested that combining amylin receptor agonism with GLP-1based therapies produces greater reductions in food intake and body weight than either approach in isolation, and early clinical data from the CagriSema programme (cagrilintide combined with semaglutide) has provided some supporting evidence in humans

Alterations in the gut flora can significantly affect brain function by modulating immune responses, producing neuroactive compounds, and affecting the integrity of the gut barrier (Zhang et al., 2022

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Several mechanisms explain why semaglutide may cause headaches

Sim R, Hernndez C