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1) are dysregulated in response to the GDH2 gene deletion, or these pathways are unable to generate sufficient glutamate inside the cells and the large pool of free NADP + required to overcome the thermodynamic barrier inherent in using the still present Gdh3, which is usually an anabolic enzyme with a higher affinity for -ketoglutarate than for glutamate

PMID: 842646 Kawashima K et al (1988): Direct determination of acetylcholine release by radioimmunoassay and presence of presynaptic M1 muscarinic receptors in guinea pig ileum

BMJ Open 13 , e072110 (2023)

[DOI] [PubMed] [Google Scholar] 26.Jensen EP, et al

Research interest includes telomerase-activity studies, circadian-rhythm research models, antioxidant-pathway studies, and long-term cellular research