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BPC-157 demonstrates a distinctive modulatory effect on nitric oxide levels, increasing or decreasing NO production as needed to maintain vascular homeostasis, always combined with counteraction of free radical formation
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Citation: [PDF] Epitalon increases telomere length in human cell lines through telomerase upregulation or ALT activity - ResearchGate
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Central to its mechanism is the modulation of the nitric oxide (NO) system, where BPC-157 has been shown to interact with both the NO synthase and NO-related pathways, contributing to its protective effects on vascular endothelium and tissue repair processes