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This may lead to reduced efficacy in regulating blood glucose levels, heightened discomfort within the gastrointestinal tract, and inhibited progress toward weight management objectives

The shorter time since market approval could explain the fewer psychiatric AEs

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GLP-1 and analogs increase glucose consumption in mature cells whereas they reduce it in progenitor cells and ADSCs, which can lead to decreased production of ATP and energy deficiency

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These findings highlight the importance of selecting the most appropriate GLP-1 agonist for individual needs and goals