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Immune homeostasis dysregulation and autoimmunity risk: as CUL5 is a key negative feedback regulator of both T-cell receptor (TCR) and cytokine signaling pathways (21), its excessive inhibition may lead to hyperactivation of T-cell signaling, breaking immune tolerance and increasing the risk of attacking normal tissues, thereby potentially triggering autoimmune diseases or severe inflammatory responses

patients with AD had a significantly lower risk for mortality than those without AD

Around 13% of American adults have used one at some stage in their lives

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