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Importantly, MEDI4166 was able to induce glucose-stimulated insulin secretion in INS1 -cells expressing endogenous GLP-1R (Supplementary Fig

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No congenital malformation (112), deficits in social interaction, anxiety-related behavior, or learning and memory deficits (115) were observed in rodent offspring that were exposed to valpromide in utero

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109 Moreover, NAD + -consuming enzymes, such as SIRT3, can also manipulate the cellular redox status via regulating the activity of enzymes for ROS generation and antioxidant factors for ROS eradication