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Diabetic painful neuropathy has been shown to be mediated via an inflammatory chemokine pathway (23) and strategies to reduce this inflammation, including gene transfer of soluble TNF receptor, have been shown to alleviate pain (24)

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To specifically dissociate the role of the fear odor-responsive neuronal ensemble from general nociceptive processing, we employed a TRAP2 strategy to selectively target 2MT-activated APC inhibitory neurons

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