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Hence, modestly diminished GLP-1 action in diabetic subjects does not likely contribute to the defective glucose-stimulated glucagon suppression that remains a characteristic of diabetic subjects 3
The mechanisms of action of GLP-1R in DM and AD overlap, which suggests that GLP-1R may be a bridge between these two diseases
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It also stimulates the transcription factor CREB, increasing insulin gene expression and biosynthesis to maintain secretory capacity (Zheng et al., 2024)
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