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Cell death is an important determinant of graft survival [11], and research on the related mechanisms has increased in recent years [12]

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Inhibition of VEGF expression through blockade of Hif1 alpha and STAT3 signaling mediates the anti-angiogenic effect of melatonin in HepG2 liver cancer cells

Currently, there are limited therapeutic options for the disease because of limited understanding of its molecular mechanism

In homozygotes, microglial and astrocyte activation appeared at 12 months, neuronal loss in the hippocampus and entorhinal cortex at 24 months, and synaptic loss at 8 months

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