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Gain-of-function CCND1 mutants are found in clonal expansions in human esophagus 29 consistent with depletion of the corresponding gRNAs in the screen

Statements Data availability statement The raw data supporting the conclusions of this article will be made available by the authors, without undue reservation

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CDK1 directly binds and phosphorylates the S447 site of ACSL4, promoting the ubiquitination degradation of ACSL4, while genetic and pharmacological blocking of CDK1 enhances ACSL4 activity, promoting ferroptosis of colon cancer cells and increasing their susceptibility to oxaliplatin