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The second direction aims to enhance HSV-1s anticancer efficacy through interference with tumor cell metabolism (17), induction of autophagy (18, 19), improvement in immune recognition processes (2023), and alteration of the tumor microenvironment itself (6, 24, 25)

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This extraordinary potency difference stems from Dihexas mechanism as an HGF mimetic binding to c-Met receptors, which triggers intracellular signaling cascades distinct from BDNFs TrkB receptor pathway

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they may be able to provide you the out-of-pocket cost as well