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The failure of macrophages to clear apoptotic cells contributes to prolonged inflammation, as these unengulfed cells can undergo secondary necrosis, releasing inflammatory mediators that exacerbate tissue damage and promote a new cycle of inflammation (134, 135)

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The key consideration is that Dihexa's effects persist beyond compound clearanceshifting protocols mid-study doesn't create an immediate reset but rather modulates ongoing neuroplastic processes already in motion

For the calculation, 300 structures were randomly extracted from the first two major clusters of both complexes