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doi: 10.1016/j.mehy.2015.09.026 51 ZhouRYazdiASMenuPTschoppJ

HepG2 cells were treated with 5 mol/ml (Figure 1A) and 10 mol/ml (Figure 1B) of ASA for 48 h and GSH levels in the mitochondria and post mitochondrial supernatant (PMS) were measured by enzymatic method as described in the Materials and Methods

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"The effect of glutathione and N-acetylcysteine on lipoperoxidative damage in patients with early septic shock." Am J Respir Crit Care Med

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Key mechanisms of action include: Fibroblast activation Suppression of collagen-degrading enzymes Reduced inflammatory cytokines Enhanced granulation tissue formation Improved angiogenesis In ischemic wound models, injectable GHK-Cu restored fibroblast function and significantly accelerated skin repair compared to controls