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Collins L, Costello RA

In AP, mitochondrial dysfunction, including ATP depletion, mtROS overproduction, and loss of m, promotes nuclear activation of stress-responsive transcriptional programs that enhance cytokine and chemokine expression and thereby contribute to pancreatic inflammation ( ATF4 is a key effector of the integrated stress response that connects mitochondrial stress, ER stress, and redox imbalance to nuclear transcriptional reprogramming

doi: 10.3390/ijms21165904

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Under this condition, almost all SCs in neighboring control HFs were tdTomato positive, while the de novo formed SCs in the ablated HFs were all tdTomato negative (Figure 2E)

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