Metabolic syndromes can increase a persons risk of cardiovascular disease, stroke and type 2 diabetes
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Our present observations are in keeping with our previous findings (17,30,31) indicating that the production of free radical, enhanced lipid peroxidation and an impairment of antioxidizing enzyme activity, and the increased generation of cytokines, such as IL-1 and TNFalpha, contribute to the pathomechanisms of stress-induced gastric lesions
