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Endocrine side-effects of anti-cancer drugs: mAbs and pituitary dysfunction: clinical evidence and pathogenic hypotheses

You might be wondering why we keep mentioning FDA approval

van der Meer 12 (1), 119133

The mechanism of this combined effect appears to be sequential: TB-500 mobilizes and migrates repair cells to the injury site (via actin regulation and chemotactic signaling), while BPC-157 creates the optimal local microenvironment (via angiogenesis, growth factor expression, and NO modulation) for those cells to perform effective repair once they arrive

[DOI] [Google Scholar] Bai Q

Zhang Z, Takahashi S, Saito S, Koizumi T