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By facilitating the entry of fatty acids into mitochondria via the carnitine palmitoyltransferase 1 (CPT1) complex, it stimulates their oxidation and causes a decrease in cell volume
These findings suggest that exogenous vitamin D is sufficient to inhibit cardiomyocyte proliferation, but the endogenous signaling pathway mediated by vitamin D receptor is not necessary for neonatal cardiomyocyte cell cycle exit in vivo
Maurer, C
10.2210/pdb2hbs/pdb 41 HayashiS.AbdulmalikO.PeranteauW
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