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p 0.05, was considered insignificant)

NSAIDs blunt thiazide response

Pei et al (64) revealed a close relationship between pathological injury of renal tissues following renal ischemia-reperfusion, increased GPX3 expression (a marker of oxidative stress) and various processes (including GSH metabolism, oxidative stress pathways and regulation of T-cell activation)

The resulting suboptimal homeostasis of GSH results in a decreased capacity to minimize oxidative stress, which is associated with poor outcomes during ageing and in multiple disease states

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