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Excessive production of ROS, largely arising from mitochondrial oxidative phosphorylation, is a major driver of oxidative damage and mitochondrial dysfunction processes that correlate with MDD symptom severity, illness chronicity, cognitive impairment, and poor antidepressant response [138, 139]

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The duration of N-acetylcysteine treatment is determined by the type of ingestion and the presence or absence of elevated serum Alanine Aminotransferase (ALT) concentrations