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The consequences of this difference have been recognised in the investigation of the toxic mechanisms associated with gout, where the concentrations of UA that induce peripheral microcrystallisation and its related inflammatory reaction are not easily reached in rodents

Oral mucosal melanin hyperpigmentation secondary to disease and physiological oral mucosal melanin hyperpigmentation may be clinically and histopathologically similar, and in some cases, it is almost impossible to determine whether oral mucosal melanin hyperpigmentation is physiological or is induced by smoking, by drugs, or by both

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Beyond Glutathione Hence, this project could be extended in the future by including some of the other molecules involved in maintaining redox homeostasis