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Esta disociacin entre efecto lipoltico y efecto sobre la glucosa e IGF-1 es la base del inters experimental del pptido

While active IGF-IR can compensate EGFR inhibition, activation of the EGFR pathway also contributes to anti-IGF-IR drugs resistance in cancer [174, 175]

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It also directly acts as a nucleophile, scavenging reactive oxygen and nitrogen species, and can modulate redox-sensitive transcription factors that regulate inflammatory and detoxification genes