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This framework provides a mechanistic explanation for why tumorsparticularly those operating near the limits of redox tolerancecan become dependent on CoQ 10 redox cycling as a last-mile defense against lipid peroxidation

& Kawamura, S

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demonstrated that, under physiological conditions, macrophages could, uptake native Hb without the binding protein Hp through CD163 (Schaer et al., 2006)

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Glutathione depletion switches nitric oxide neurotrophic effects to cell death in midbrain cultures: implications for Parkinsons disease