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By performing acute A applications ex vivo , the authors proposed that soluble A might trigger the decrease of synaptic plasticity in neocortical pyramidal cell networks during early stages of AD pathogenesis by preferentially targeting postsynaptic AMPA receptors (Shemer et al., 2006)

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Inhibition of Leukotriene B4-Induced CD11B/CD18 (Mac-1) Expression by BIIL 284, a New Long Acting LTB4 Receptor Antagonist, in Patients With Rheumatoid Arthritis