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The mechanism is through a variety of autophagy-related proteins (including Beclin1 and LC3B, which are the initial protein and structural components of autophagosomes, respectively) to form double membrane vesicles to engulf damaged proteins or organelles and bind to lysosomes [31]

This therapeutic gap necessitates exploration of alternative strategies targeting the underlying inflammatory drivers of disease progression

Slow Digestion: Delaying gastric emptying to stabilize blood sugar

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Fibroblasts are the cells responsible for producing collagen and other extracellular matrix components that form the structural basis of fascia