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TREK-1 channels are "leak" channels that maintain resting membrane potential and regulate neuronal excitability

7b), confirming our hypothesis

ManducaABaraALarrieuTLassalleOJoffreCLaySet alAmplification of mGlu5-endocannabinoid signaling rescues behavioral and synaptic deficits in a mouse model of adolescent and adult dietary polyunsaturated fatty acid imbalance

Shu DY, Butcher E, Saint-Geniez M

NADPH depletion (from glutathione recycling overload, G6PD deficiency, or high oxidative stress) limits NQO1 throughput Selenium is required for glutathione peroxidase, which works alongside NQO1 to manage oxidative stress

Thats why many experts consider building it endogenously via NAC and glycine a more sustainable route