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Its overactivation disrupts nicotinamide metabolism, reduces NAD+ levels, promotes hepatic lipid accumulation and lipotoxicity, and represents an important therapeutic target for hepatic metabolic disorders

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10.1016/j.heares.2012.08.002 57 VuA

The function of the AT 2 receptor is less well understood, possibly because the AT 2 receptor has a low degree of expression compared with that of the AT 1 receptor