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Gastrointestinal Conditions That Limit GLP-1 Eligibility GLP-1 medications slow gastric emptying as part of their weight loss mechanism, which creates challenges for patients with certain digestive disorders

With the increase of FFA oxidation, PPAR is activated, and the expression of BBD and OCTN2 in oxidative tissue is simulated to facilitate disposal (85)

But not everyone can or wants to use these medications, which has led to interest in natural alternatives that might boost GLP-1 levels or mimic its effects

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Collectively, these hormones regulate appetite, glucose homeostasis, and whole-body energy balance, while also influencing central neuroendocrine processes