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During inflammatory bowel disease, colonic cells ramp up PepT1 expressioncreating disease-specific targeting in research models

Although the transfer of metabolic substrates form Schwann cells to axons was reported about two decades ago (Vega et al., 2003), the specific mechanism behind the metabolic transfer in the PNS is still unclear

We found that this was not mediated by B-cell death induced by apoptosis, but rather by the transition into a hyporesponsive state

GHK-Cu is particularly relevant for this population because of its documented ability to remodel UV-damaged collagen and activate the skin's antioxidant defenses against ongoing solar damage