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doi: 10.1093/cid/civ933 44 PappasP

[119] designed an HCS peptide SP9 that can specifically interrupt membrane fusion triggered by Ca 2+ via disturbing the primary interface between SNARE and the Ca 2+ -binding C2B domain of Syt1 (Figure 7)

GLP-1R agonists with prolonged half-life (generated by amino acid substitutions and linkage to a fatty acid, the Fc region of IgG4 or albumin [16]) have been developed to treat type 2 diabetes [17]

Commercial formulations of hydroquinone contain the preservative metabisulphite, which can cause an allergic reaction

The in vivo rat wound-chamber data, in which injected GHK-Cu increased connective-tissue accumulation in a concentration-dependent way while an inactive control peptide did not, are the systemic-route corroboration of these culture findings and are important because they show the effect surviving the transition from a dish to a living animal

Supports the creation of energy within the heart muscle*