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During the resolution of inflammation, SPMs produced by polymorphonuclear cells and macrophages act to limit local tissue damage caused by inflammatory responses, including signaling cessation of polymorphonuclear cell infiltration, macrophage switching to an anti-inflammatory phenotype, apoptotic cell clearance, and reducing pain caused by nociceptor sensitization (49, 61)

Hepatitis B virus (HBV) infection can progress to life-threatening liver fibrosis, cirrhosis, and hepatocellular carcinoma (HCC)

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Standard approaches for soft tissue injuries include NSAIDs, corticosteroid injections, physical therapy, and in severe cases, surgery

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