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Pulmonary fibrosis is characterized by the accumulation of myofibroblasts, the activated form of fibroblasts, which are responsible for the pathogenic synthesis of collagen and the deposition of the extracellular matrix in the alveolar structures (Mostafa et al., 2022)

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The cytokine expression in peripheral blood mononuclear cells after trickle infections was polarized more towards a Th2 response with a gradual increase in the Th1 profile in the chronic disease stage