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Studies in mouse models with heterozygous GPx deficiency reveal heightened susceptibility to oxidant stress, emphasizing its protective function in heart tissues

the vast majority is stored inside cells and bones

SIRT1 induces the circadian transcription of core clock genes, such as Cry1, Per2, Ror, and Bmal1 via either rhythmically deacetylating BMAL1 or PER2

When your antioxidant defenses are low, your body struggles to neutralize harmful free radicals, which damages cells and interferes with important processes, including sleep regulation

Therefore, targeted activation of antioxidant systems, especially the GSH system, is an essential treatment strategy to protect cardiomyocytes against oxidative damage

Without independent groups confirming these findings in diverse populations, the evidence base remains narrower than ideal