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Bolton WD, Hofstetter WL, Francis A, Correa AM, Ajani, JA, Bhutani B, Erasmus JJ, Komaki R, Maru D, Mehran RJ, Rice DC, Roth JA, Swisher SG , Vaporciyan AA, Walsh GL, Weaver J, Rhines L

Seachrist, Darcie D., et al

The metabolism of homocysteine: pathways and regulation

2 These include: A ME/CFS-like phenotype with post-exertional malaise and impaired bioenergetics A dysautonomia/POTS-dominant presentation A mast celldriven inflammatory phenotype Persistent cardiopulmonary injury This heterogeneity explains why a single treatment approach fails

The entire RNA genome of four independent plaques was sequenced and only one type of mutation was observed (Figure 3A)