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Clinical alterations resulting from these mutations are hypothesized to be derived from either loss or gain of function of GCase and interaction with its substrates like glycosphingolipids (GSLs), glucosylceramide (GlcCer) and glucosylsphingosine (GlcSph)

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While essential for survival during acute stress, sustained high cortisol disrupts multiple systems, including digestion and nutrient absorption

Acceleration of brain amyloidosis in an Alzheimers disease mouse model by a folate, vitamin B 6 and B 12 -deficiency diet

Experimental & Molecular Medicine, 52, 1496516

Deviations from these conditions will shorten shelf life, sometimes dramatically