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Pretreatment of HaCaT cells with an IP3R inhibitor indicates that GSK3 may regulate the activation of IP3R, and thereby modulate the release of Ca 2+ from the endoplasmic reticulum into the cytoplasm, thus indirectly regulating mitochondrial damage, and apoptosis

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variabilis , SOD activity increased from 46.08 USOD mg -1 protein min -1 in the control to 73.42 USOD mg -1 protein min -1 at 300 g L -1 Pb and to 142.08 USOD mg -1 protein min -1 at 500 g L -1 Pb, corresponding to relative increases of 59.3% and 208.3%, respectively

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