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As a major intracellular antioxidant, it directly neutralises reactive oxygen species (ROS) and free radicals that accumulate during normal metabolism and are elevated in fatty liver disease

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When activated by ROS or electrophilic substances, Nrf2 exits Keap1 and moves to the nucleus, functioning as a transcription factor to enhance the production of subsequent antioxidant and detoxi-fying enzymes like HO-1, NAD (P)H, NQO-1, SOD, and GSH-PX (112, 113)