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Based on the above research evidence, the study hypothesis is that supplemeting LA appropriately to combat MASLD may be effective

Glutathione-S-transferase M1 and T1 null genotypes are associated with hypertension risk: A systematic review and meta-analysis of 12 studies

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Theoretically, chronic lipolysis activation could deplete essential lipid stores or disrupt adipose tissue-derived signaling pathways (via adipokines), though this remains speculative

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Sulforaphane alone cannot directly stimulate autophagy at the 50 mg/kg dose associated with neuroprotection, but it could rescue rotenone-inhibited autophagy (Fig