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The ER mediates oxidative protein folding, necessary for correct protein functioning, and the formation of disulfide bridges is mainly catalysed by PDI, encoded by P4hb , which functions as an electron acceptor and transfers the electrons indirectly to oxygen, resulting in H 2 O 2 production 10

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However, as outlined below, the molecular defects described to date are not sufficient to explain disease in many EDS patients, including those with the most common classical and hypermobility types

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Siddiqui WA, Ahad A, Ahsan H (2015) The mystery of BCL2 family: Bcl-2 proteins and apoptosis: an update