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Intestinal dysbiosis is characterized by a reduction in the flora producing anti-inflammatory metabolites (such as short-chain fatty acids and secondary bile acids) and their encoding genes, along with an enrichment of flora producing the pro-inflammatory metabolite trimethylamine, resulting in metabolic imbalance

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Cellular Repair and Tissue Recovery GH and IGF-1 signaling influences cellular repair processes, including collagen synthesis, wound healing, and tissue regeneration