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This scenario could explain the rapid need to eliminate the unbound form of these proteins, as potentially facilitated by the 20S proteasome in this context

doi: 10.1007/s11010-016-2703-4 92 AlhibshiAHOdawaraASuzukiI

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Similarly, the small molecule activator 2-APQC effectively enhances SIRT3 activity, alleviating cardiac hypertrophy and myocardial fibrosis induced by isoproterenol (ISO)