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In the IL-13 model, mice treated with IL-13 and GGsTop exhibit a lung inflammatory response similar to that of mice treated with IL-13 alone

This is partly due to the poor logistics associated with sterilization of high glucose containing alkaline additives, a process that requires separation of the high sugar fraction from the alkaline component to prevent caramelization of the former at high temperature

These subcellular alterations not only directly damage structural cells (e.g., airway epithelium) but also release damage-associated molecular patterns (DAMPs) and oxidized lipids that activate and recruit immune cells (85)