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Moreover, in models of ischemiareperfusion injury and in KEAP1-inactivated lung cancer, inhibition or genetic ablation of CoQ biosynthetic enzymes restored sensitivity to ferroptosis or radiotherapy 115,116 , underscoring the reliance of some cancer cells on an abundant CoQ pool for redox defense and survival

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4b, c, d), accompanied by reduced expression of macrophage markers, including Cd68 and Itgam across all three adipose depots (Supplementary Fig

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