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Similarly, pharmacological inhibition of HSL and knockdown of PNPLA2 reduced the expression of known PPAR target genes in which LDs were shown to be a source of PPAR and PPAR ligands that could be generated within minutes of stimulating lipolysis (3)

doi: 10.1016/j.jclinepi.2020.08.005 252

Drug-induced oxidative stress and toxicity

The peptides hypothetical binding capability may contribute to the possible hindering of cellular uptake of OxLDL, a complex that appears to have proatherogenic properties

doi: 10.3390/foods12163086 34 AminnezhadSZonobianMAMoradi DoukiMMohammadiMRAzarakhshY