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The observed reduction in GSSG levels reflects a compensatory regulation of redox balance, suggesting a potential self-protective mechanism that may play a crucial role in maintaining neurological homeostasis [48, 49]

SIRT3 is indispensable for maintaining mitochondrial protein acylation homeostasis, and thereby cell metabolism, through deacetylating key enzymes in various metabolic pathways [28, 53]

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Importantly, there were no cardiovascular adverse events, no clinically significant changes in blood pressure or heart rate, no alterations in thyroid function, no immunogenicity with no anti-AOD-9604 antibodies detected, and no signs of tolerance or receptor desensitization