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The postulated mechanism for causing such attacks is through stimulation of the porphyrin precursor, delta-aminolevulinic acid (ALA) Laboratory tests: Diclofenac prolongs platelet aggregation without affecting the bleeding time, plasma fibrinogen and factors V and VII to XII
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To correlate lux-based bioluminescence and bacterial metabolic activity, stationary-phase JE2 and JE2-lux cultures were subcultured at 1:200 in MHB, incubated at 37 C for 3 h with shaking, then treated with 00.5 mM sodium arsenate (Sigma) for 30 min
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