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Genes 12 , 796 (2021)

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Cardiac cells that have been genetically engineered to lack NRF2 have been shown to be unable to resist damage due to reactive oxygen radicals, presumably due to a deficiency of SOD (3)

doi: 10.1530/JOE-13-0221

Moreover, preliminary research has indicated that alterations in thiol levels during oxidative stress may be associated with the size of the infarct resulting from ischemic stroke

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