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Slow replication fork velocity of homologous Recombination-Defective cells results from endogenous oxidative stress

Research models reveal that senescent cells accumulate with aging and in response to various stressors, secreting inflammatory factors known as the senescence-associated secretory phenotype (SASP) that promotes chronic inflammation and can induce senescence in normal cells

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[5] By simultaneously providing all these B vitamins, the injection ensures that the body has all necessary co-factors in adequate supply

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