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The mechanisms include blood pressure reduction, improvement in lipid profiles, anti-inflammatory effects on the arterial wall, and possible direct cardioprotective effects through GLP-1R expressed on cardiomyocytes
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J.HateleyC
This mechanism has been validated in clinical trials
Our work documented the ability of site-specific acetylation to increase TrxR1 activity by reducing oligomerization (Wright et al., 2018), and here we found acetylation also provided robust resistance to oxidative damage and peroxide-induced TrxR1 oligomer formation
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